Specificity-of-AD7C-NTP
7页1、Volume 1998, Article 4A June 1998 The MIT Press Journal of Contemporary Neurology ISSN 1081-1818. MIT Press Journals, Five Cambridge Center, Cambridge, MA 02142, USA; tel.: (617) 253-2889; fax: (617) 577-1545; journals- ordersmit.edu, journals-infomit.edu. Published one article at a time in html and PDF source form on the Internet. For more information and other articles see: http:/mitpress.mit.edu/CONE/ 1998 Massachusetts Institute of Technology. Subscribers are licensed to use journal articles
2、 in a variety of ways, limited only as required to insure fair attribution to authors and the Journal, and to prohibit use in a competing commercial product. See the Journals World Wide Web site for further details. Address inquiries to the Subsidiary Rights Manager, MIT Press Journals; (617) 253-2864; journals-rightsmit.edu. 2Contemporary NeurologyVolume 1998, Number 4A Specificity of AD7C-NTP as a Biochemical Marker for Alzheimers Disease Hossein Ghanbari, PhD Kasra Ghanbari Michael Munzar, MD
3、 Paul Averback, MD From the Nymox Corporation, Rockville, Maryland Address reprint request and correspondence to Hossein A. Ghanbari, PhD, Nymox Corpora- tion, 5516 Nicholson Lane, Suite 100A, Rockville, MD 20895, IThe 41-kD protein AD7C-NTP is present in neurons. It is selectively upregulated in the Alzheimers disease (AD) brain and is associated with the pathology of the dis- ease. In situ hybridization and immuno- staining studies have localized AD7C-NTP gene expression in neurons. Overex- p
4、ression of AD7C-NTP in transfected neu- ronal cells promotes neuritic sprouting and cell death. Using an enzyme-linked sandwich immunoassay constructed with antibodies to the recombinant protein, AD7C-NTP levels have been measured in cerebrospinal fluid samples from cases of AD as well as age-matched controls and a variety of neurological disease controls, including cases of stroke, Picks disease, amyotrophic lateral sclerosis, diffuse Lewy body disease, and certain psychiat- ric disorders of th
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